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frisbi platforms catalogue centers working groups training submit a proposal login french infrastructure for integrated structural biology platforms catalogue centers working groups training submit a proposal login home the french infrastructure for integrated structural biology ( frisbi ) provides an infrastructure for integrative structural biology approaches, from the molecular to the cellular level, integrating multi-resolution data from x-ray crystallography, small angle x-ray scattering, nmr, cryo-em and functional data including development for protein expression and crystallization. frisbi is open to structural and molecular and cell biologists from both academia and industry from france and europe . a simple, transparent reviewed process will provide access based on its feasibility and resource availability. applications for access can be submitted at any time . users will be required to contribute towards the costs of access. two of the frisbi centers are part of the european instruct-eric infrastructure the 5 th frisbi call for proposals for structural biology training courses (to be held in 2019 in france) is now closed. acknowledgement : in published material, please acknowledge the use of frisbi infrastructure, expertise using the following text: this work was supported by the french infrastructure for integrated structural biology (frisbi) anr-10-inbs-05. a copy of the communication / publication (pdf) should be sent to frisbi article "biologie structurale: des équipements de pointe à disposition de l'industrie". read more (970.0 kb) latest news 2017, a year with the cnrs in alsace 2018-09-13 coming soon in english more info all news transcription defects related to the xpc factor implicated in the disease of children of the moon 2018-07-04 children of the moon cannot be exposed to the sun because of their deficiency in dna repair proteins including xpc, the factor that recognizes ultraviolet damage. however, this failure is not enough to explain all their symptoms. in this study, researchers from f... more info all news transcription and intellectual disability: a mutation resulting in the almost complete loss of a general transcription factor has a much less severe effect than expected 2018-04-13 the transcription is an essential mechanism for gene expression and is regulated by many protein factors. in humans, the tfiid factor, composed of the tata box binding protein and 13 associated factors (tafs: tbp-associated factors), is crucial to the initiation of transcription by rna polymerase ii. in this study, laszlo tora’s tea... more info all news 2017, a year with the cnrs in alsace 2018-09-13 coming soon in english transcription defects related to the xpc factor implicated in the disease of children of the moon 2018-07-04 children of the moon cannot be exposed to the sun because of their deficiency in dna repair proteins including xpc, the factor that recognizes ultraviolet damage. however, this failure is not enough to explain all their symptoms. in this study, researchers from frédéric coin's team , including nicolas le may, (cnrs / inserm / university of strasbourg) showed that the xpc factor was also involved in a fundamental mechanism of gene expression, transcription. these results are published on july 4, 2018 in the journal nature communications . xeroderma pigmentosum is a genetic disease that can result from a mutation in the gene coding for the xpc protein, a dna damage sensor that recognizes lesions produced by ultraviolet rays. patients who are very sensitive to the sun have an increased risk of skin cancer but also develop neurological or ocular disorders. these symptoms, originally associated with defects in dna repair, may also be related to disturbances in transcription, a fundamental mechanism of gene expression. frédéric coin's team therefore sought to establish the link between the xpc factor and transcription. in normal cells, the researchers observed the presence of xpc on nearly 500 genes. the xpc occupancy sites on dna coincide precisely with those of rna polymerase ii, the enzyme that catalyzes transcription. on the other hand, in cells derived from patients in whom xpc is defective, they showed that the expression of these genes was deregulated and that rna polymerase ii was no longer recruited correctly on their promoter, thus highlighting the link between the xpc factor and transcription. the researchers then analyzed histone modifications that are essential to the development of an ideal chromatin environment for gene expression. they were particularly interested in the acetylation of histone h3, mediated by two major transcription complexes, saga and atac. the researchers observed that the acetylation of histone h3 in the 500 genes targeted by xpc was deficient in its absence. this is explained by the fact that xpc interacts with the atac complex and allows its recruitment on genes. finally, they showed that the xpc protein was specifically recruited from the 500 genes through its interaction with the e2f1 protein, a transcription factor recognizing particular dna sequences present upstream of the promoters of the 500 genes. thus a succession of events, initiated by the recruitment of the transcription factor e2f1 followed by the arrival of xpc and the recruitment of atac on specific genes leads to the remodeling of chromatin and the expression of these genes. these results establish that in addition to repairing dna, xpc regulates transcription, and provide a better understanding of the molecular basis of defects in patients with xeroderma pigmentosum. this study was funded by anr, the cancer research association, korea's national research foundation, the national league against cancer, the foundation for medical research, and the south province research incentive award in new caledonia. transcription and intellectual disability: a mutation resulting in the almost complete loss of a general transcription factor has a much less severe effect than expected 2018-04-13 the transcription is an essential mechanism for gene expression and is regulated by many protein factors. in humans, the tfiid factor, composed of the tata box binding protein and 13 associated factors (tafs: tbp-associated factors), is crucial to the initiation of transcription by rna polymerase ii. in this study, laszlo tora’s team at the igbmc (cnrs, inserm and university of strasbourg) associated with american, british and australian collaborators studied the consequences of a mutation in the gene coding for one of the associated factors, the taf8 protein, in a child with an intellectual disability. although this mutation causes an almost complete loss of taf8, the researchers showed that it did not affect the overall transcription, contrary to what is observed after total inactivation of the taf8 gene in mice. these results were published on april 13 in the journal human molecular genetics. the expression of genes coding for proteins requires the assembly of many molecules on a sequence promoting transcription. the first complex to bind to the promoter is the general transcription factor, tfiid, composed of the tata box binding protein (tbp) and 13 associated factors (tafs). current studies suggested that the assembly of an entire tfiid complex was essential for the initiation of transcription and survival of eukaryotic cells. in a child with an intellectual disability and a major developmental delay, laszlo tora’s team showed that a mutation of the taf8 gene, one of the associated factors, results in a shift in the genetic code reading frame and in the production of an extremely unstable mutant protein, undetectable in cells derived from the patient. in addition, immunoprecipitation and proteomic analyses show that, in these cells, the formation of the tfiid complex is strongly altered and that only partial complexes are identified. eventually, the researchers showed that this disorganization of the tfiid complex does not affect the

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Language: fr
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